Case 2: Gingival Recession and Abnormal Lucencies

Patient Presentation[^9]

  • Chief Complaint: The patient is primarily concerned with the aesthetic appearance of their gingival recession.
  • ==Symptoms:==
    • No pain or discomfort reported.
    • No history of trauma.
  • ==Clinical Findings:==
    • ==Vitality: All lower anterior teeth tested vital.==
    • ==Caries/Fractures: No evidence of caries or tooth fractures.==
    • ==Periodontal Status: Minor periodontal bone loss and presence of calculus on enamel surfaces.==

==Radiographic Assessment==

  • ==View: PA of the anterior mandible (showing teeth 43 to 33).==
  • ==Findings:==
    • ==Lucencies: Presence of radiolucencies at the periapical regions of teeth 32, 31, and 42.==
    • ==Lesion Characteristics: The lesions involving 32 and 31 appear as one continuous lobular lesion, while there is a solitary lesion at the apex of 42.==
  • ==Differential Diagnosis: Inflammatory lesions in this area are typically associated with caries, fractures, or other coronal issues.==

Radiographic Findings

Identify and describe the abnormal lucencies present in the image:

  • List at least three distinct radiographic features.
    • Not completely radio opaque; ==presents as decreased density (pinpoint areas) in the periapical region of the lower anterior teeth (specifically around teeth 32 and 31).==
    • Most likely Cemento osseous dysplasia (COD). The bone is not eroded, but the density is altered.
    • Radiographic bone loss (minor periodontal bone loss).
    • Calculus on enamel surfaces.
  • ==Progression/Maturation:==
    • ==These lesions can mature over time, transitioning from radiolucent to radiopaque as they form abnormal, brittle bone.==
    • Once the lesion is mature (opaque), it typically stops growing.

Diagnostic Considerations

  • Provide a differential diagnosis (ddx).

    • Periapical granuloma. Vitality testing is the most important step to rule out pulp necrosis/inflammatory lesions caused by blunt trauma or caries. Inflammatory lesions usually involve bone erosion, whereas COD may just show decreased density initially.
    • Cemento osseous dysplasia (still differential because it has to be confirmed).
      • ==Epidemiology: Extremely common, particularly in Australia/Perth.==
      • ==Demographics: More prevalent in females and individuals with African, Asian, or Mediterranean backgrounds.==
      • ==Age: Typically an “older person’s disease”; if seen in a young child, COD can likely be ruled out.==
  • Determine if further imaging is necessary.

    • Another PA should be taken after 2 years to see if the COD lesion has matured or is still active.
    • If the lesion remains stable or has matured (become opaque) after two years, no further radiographs are needed unless the tooth becomes symptomatic.
    • When it matures it looks more radiopaque (not radiolucent).

==Management and Follow-up==

== Clinical Management==

==Treatment: No active treatment or extirpation is required.==

  • ==Warning: Avoid unnecessary intervention, as “infected COD” can involve the entire bone.==
  • ==Diagnosis: A confirmed diagnosis requires histology, which is unnecessary as the condition is asymptomatic and requires no management.==