Pediatric Important Numbers
How to use this note
A quick-reference consolidation of every quantitative fact from the DMD3S1 Pediatric Dentistry lectures and workshops, ordered by exam priority: 1) caries statistics → 2) local anaesthesia → 3) medications → 4) other statistics.
- Every figure links to its source lecture via wikilink.
- Where a figure comes from an in-lecture paper, that paper is named in plain text in the Paper column (these are not vault notes).
—= stated by the lecturer without a cited source.- Drug doses cross-reference
[[R1 Antibiotic Selection Table]]for penicillin-allergy tiers.
1. Caries Statistics
Highest-yield caries figures
= Primary→permanent caries risk ≈ 3× (RR 2.6). MIH ≈ 1 in 7 children; HSPM ≈ 1 in 10.
Caries risk, detection & ICDAS
| Statistic | Figure | Source Lecture | In-lecture Paper |
|---|---|---|---|
| Moderate caries-risk threshold | 1–2 new lesions/year | L1 The Paediatric Clinical examination LMS | — |
| High caries-risk threshold | ≥3 new lesions/year | L1 The Paediatric Clinical examination LMS | — |
| Bitewing = gold standard for caries; film size for young child | Size 0 | L1 The Paediatric Clinical examination LMS | — |
| Occlusal lesions detected by bitewing (sensitivity) | ~33% | W1 ICDAS and intro to caries | — |
| ICDAS second-digit severity range | 0–6 | W1 ICDAS and intro to caries | — |
| ICDAS codes favouring preventive/microinvasive care | 0–4 | W1 ICDAS and intro to caries | — |
| ICDAS codes indicating operative treatment | 5–6 | W1 ICDAS and intro to caries | — |
| ICDAS threshold used in epidemiological/field surveys | 3+ | W1 ICDAS and intro to caries | — |
ICDAS code key (second digit): 0 sound · 1 first visual change (after drying / in pit-fissure) · 2 distinct change visible when wet · 3 localised enamel breakdown / microcavitation, no dentine · 4 dark dentine shadow ± enamel breakdown · 5 distinct cavity, dentine visible, <½ surface · 6 extensive cavity, dentine visible, >½ surface. — W1 ICDAS and intro to caries
Fluoride
| Statistic | Figure | Source Lecture | In-lecture Paper |
|---|---|---|---|
| Recommended fluoride toothpaste (prevention & sensitivity) | 1000 ppm | L5 MIH and HSPM, W1 ICDAS and intro to caries | — |
| Commercial “3–5 year” children’s pastes (often only) | 500 ppm | W1 ICDAS and intro to caries | — |
| Toothbrushing frequency | Twice daily | W1 ICDAS and intro to caries | — |
Caries as a predictor / progression
| Statistic | Figure | Source Lecture | In-lecture Paper |
|---|---|---|---|
| Children with MIH — odds of DMFT > 0 vs non-MIH | 5.89× (95% CI 2.69–12.88; P<0.05) | W1 ICDAS and intro to caries | Americano et al. 2016 |
| Primary tooth with proximal caries → caries on adjacent proximal surface | 69% | W1 ICDAS and intro to caries | Dean et al. 1997 |
| Proximal lesion in one quadrant → another primary-molar proximal lesion in a different quadrant | 89% | W1 ICDAS and intro to caries | Dean et al. 1997 |
| Caries in primary teeth → relative likelihood of permanent-tooth caries | ≈3× (RR 2.6; 95% CI 1.4–4.7) | W1 ICDAS and intro to caries | Li and Wang 2002 |
| Predictive value of primary-molar caries for permanent-dentition caries | 85.4% (highest) | W1 ICDAS and intro to caries | Li and Wang 2002 |
| ↑ risk of mesial caries on first permanent molar from distal caries on second primary molar | 15× | W1 ICDAS and intro to caries | — |
Note:
[[W3 Extra information]](caries-prediction workshop handout) is the in-vault source for the Americano, Dean, and Li & Wang figures above.
MIH prevalence (developmental, caries-relevant)
| Statistic | Figure | Source Lecture | In-lecture Paper |
|---|---|---|---|
| MIH prevalence (population-dependent) | 3% – 25% | L5 MIH and HSPM | — |
| MIH worldwide range | 2.4% – 40.2% | L5 MIH and HSPM | Jälevik, 2010 |
| MIH prevalence — Australia | 22% – 44% | L5 MIH and HSPM | Arrow et al. 2008; Balmer et al. 2005 |
| MIH prevalence — New Zealand | 14.9% – 18.8% | L5 MIH and HSPM | Mahoney & Morrison, 2009/2011 |
| Pooled global MIH prevalence (meta-analysis, 70 studies) | 14.2% (≈1 in 7) | L5 MIH and HSPM | Zhao et al. 2018 |
| MIH — males / females | 14.3% / 14.4% (NS) | L5 MIH and HSPM | Zhao et al. 2018 |
| MIH — ≤10 yrs / >10 yrs | 15.1% / 12.1% | L5 MIH and HSPM | Zhao et al. 2018 |
| MIH — South America / Spain (highest) | 18.0% / 21.1% | L5 MIH and HSPM | Zhao et al. 2018 |
HSPM prevalence & link to MIH
| Statistic | Figure | Source Lecture | In-lecture Paper |
|---|---|---|---|
| HSPM global prevalence | ≈10% (range 5–15%) | L5 MIH and HSPM | — |
| HSPM prevalence — Australia | 14% | L5 MIH and HSPM | Owen et al. 2017 |
| HSPM as predictor of MIH (odds ratio) | 4.66 (up to 6× in some studies) | L5 MIH and HSPM | — |
| HSPM–MIH co-occurrence | ~20% | L5 MIH and HSPM | Garot et al. 2018 |
| Child with HSPM → ↑ risk of similar permanent-dentition defects | 2.4 – 10× | W1 ICDAS and intro to caries | — |
2. Local Anaesthesia
Highest-yield LA figures
==Lignocaine & Articaine max = 7 mg/kg; Bupivacaine max = 2 mg/kg.== Australian carpule = 2.2 mL. ==2% solution = 20 mg/mL; 4% = 40 mg/mL.==
Agents
| Agent | Concentration | Vasoconstrictor | Max dose | Notes | Source | Paper |
|---|---|---|---|---|---|---|
| Lignocaine | 2% (= 20 mg/mL) | Adrenaline 1:80,000 | 7 mg/kg | Standard agent; acidic infected tissue slows onset/shortens action | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Articaine | 4% (= 40 mg/mL) | Adrenaline 1:100,000 | 7 mg/kg | effective mandibular infiltration, short onset; 1:80,000 better haemostasis than 1:100,000 | L6 Antibiotics LA and Analgesics in Paediatric Dentistry, W1 ICDAS and intro to caries | — |
| Bupivacaine | 0.5% | Adrenaline 1:200,000 | 2 mg/kg | Long-acting: ~6 h pulpal, 9–12 h soft tissue → ↑ self-injury risk in children | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Topical lignocaine | 2–5% gel / 5% adhesive vs 10% ointment | — | (verify product) | Dwell ~2 min, then wipe with damp gauze; verify concentration | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
Dose calculation & technique
| Parameter | Figure | Source | Paper |
|---|---|---|---|
| Percentage → mg/mL conversion | 1% = 1 g/100 mL = 10 mg/mL (so 2% = 20 mg/mL, 4% = 40 mg/mL) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Carpule volume (Australia standard) | 2.2 mL (intl. often 1.8 mL or 1.7 mL) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| 2% lignocaine content per carpule | 2.2 mL → 44 mg · 1.8 mL → 36 mg · 1.7 mL → 34 mg | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Worked max dose, 25 kg child (2% lignocaine) | 7 × 25 = 175 mg = 8.75 mL ≈ 3.9 carpules | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Worked max dose, 18 kg child (4% articaine) | 7 × 18 = 126 mg = 3.15 mL ≈ 1.4 carpules | W1 ICDAS and intro to caries | — |
| IANB depth to bony contact | ~20 mm | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Needle gauge for IANB | 27–30 G (short needle in children) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Staged injection volume | ½ carpule initially, re-inject only if inadequate | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
Note: the 18 kg articaine worked example is sourced from the W3 abscess workshop case (
[[W3 Case 2_integrated]]).
3. Medications — Dosages & Timings
Highest-yield drug doses
Penicillin V / Amoxycillin / Cephalexin: 12.5 mg/kg (max 500 mg). Clindamycin 7.5 mg/kg (max 300 mg); Metronidazole 10 mg/kg (max 400 mg). Paracetamol 15 mg/kg (max 4 g/day); Ibuprofen 5–10 mg/kg (max 2400 mg/day).
Antibiotics — odontogenic infection (all oral, 5-day course)
See [[R1 Antibiotic Selection Table]] for the penicillin-allergy tier list.
| Drug | Used For (Condition) | Dose | Frequency / Duration | Source | Paper |
|---|---|---|---|---|---|
| Phenoxymethylpenicillin (Pen V) | Odontogenic infection — drug of first choice | 12.5 mg/kg up to 500 mg | 6-hourly × 5 days (1 h before / 2 h after meals) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Amoxycillin | Odontogenic infection | 12.5 mg/kg up to 500 mg | 8-hourly × 5 days | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Cephalexin | Odontogenic infection (penicillin-sensitive alternative) | 12.5 mg/kg up to 500 mg | 8-hourly × 5 days | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Clindamycin | Odontogenic infection — first choice if penicillin-allergic | 7.5 mg/kg up to 300 mg | 8-hourly × 5 days | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Metronidazole | Spreading / anaerobic infection (supplements penicillin or clindamycin) | 10 mg/kg up to 400 mg | 12-hourly × 5 days; avoid alcohol 24 h after course (adults) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
Suspension concentrations to specify on the script: Pen V 125 / 150 / 250 mg per 5 mL · Amoxycillin 100 mg/mL, 125 / 250 / 500 mg per 5 mL · Cephalexin 125 / 250 mg per 5 mL · Clindamycin 150 mg caps · Metronidazole 200 mg/5 mL (40 mg/mL). — L6 Antibiotics LA and Analgesics in Paediatric Dentistry
Analgesics
| Drug | Used For (Condition) | Dose | Frequency / Duration | Source | Paper |
|---|---|---|---|---|---|
| Paracetamol | Post-op / dental pain & fever — first line | 15 mg/kg (max 1 g/dose) | 4–6-hourly, max 4 g/day; conc. 120 mg/5 mL (24 mg/mL) | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | — |
| Ibuprofen | Post-op / dental pain — first line (anti-inflammatory) | 5–10 mg/kg | 6–8-hourly, max 2400 mg/day; conc. 100 mg/5 mL; ≤3 days without review | L6 Antibiotics LA and Analgesics in Paediatric Dentistry | Kent et al. J Am Coll Cardiol. 2018;72(3):255-67 |
Analgesic age restrictions
- Codeine: not in children <12 yrs; also contraindicated 12–18 yrs after tonsillectomy/adenoidectomy. Rescheduled to S4 (prescription-only) on 1 Feb 2018. — Pediatrics. 2016;138(4):e20162396
- Aspirin: banned <16 yrs (Reye syndrome). — Glasgow JF, Drug Saf 2002; Schrör K, BMJ 2002; Macdonald S, BMJ 2002
Orofacial infection medications
| Drug | Used For (Condition) | Dose | Frequency / Duration | Source | Paper |
|---|---|---|---|---|---|
| Aciclovir (PO) | Primary herpetic gingivostomatitis (HSV) — antiviral | 20 mg/kg | 5×/day; within first 72–96 h (prodromal phase); limited evidence <2 yrs | L7 Orofacial Infections In Paediatric Dentistry | — |
| Aciclovir (IV) | Severe / immunocompromised HSV | 10 mg/kg | IV | L7 Orofacial Infections In Paediatric Dentistry | — |
| Chlorhexidine 0.2% (swab) | Herpetic gingivostomatitis — severe ulceration, young children | 0.2% on cotton wool | Over affected areas | L7 Orofacial Infections In Paediatric Dentistry | — |
| Chlorhexidine 0.2% (mouthwash) | Herpetic gingivostomatitis — older children | 0.2%, 10 mL | Every 4 h | L7 Orofacial Infections In Paediatric Dentistry | — |
| Chlorhexidine gel | Post-trauma (concussion/subluxation) oral hygiene | Finger/brush application | First week only, then brush alternate days | L8 Dental Trauma In the Primary Dentition | — |
| Difflam C (benzydamine + chlorhexidine) | Herpetic gingivostomatitis — combination rinse | Benzydamine 0.15% + chlorhexidine 0.12% | — | L7 Orofacial Infections In Paediatric Dentistry | — |
| Tetracycline / minocycline mouthwash | Herpetic gingivostomatitis — children >12 yrs | — | — | L7 Orofacial Infections In Paediatric Dentistry | — |
| Varicella antivirals (aciclovir/valaciclovir/famciclovir) | Varicella (chickenpox) | — | Most effective within 24 h of rash onset | L7 Orofacial Infections In Paediatric Dentistry | — |
| Paracetamol | Herpetic gingivostomatitis — pain/fever | 15 mg/kg | Every 4–6 h | L7 Orofacial Infections In Paediatric Dentistry | — |
Topical anaesthetic cautions (infection setting)
- Viscous lidocaine 2% — not recommended (systemic overdose; child cannot spit).
- Benzocaine gel — avoid <6 yrs (methaemoglobinaemia risk). — L7 Orofacial Infections In Paediatric Dentistry
4. Other Statistics
Tooth-number & size anomalies (prevalence)
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Hyperdontia — primary / permanent dentition | 0.2–0.8% / 0.5–3.5% | L2 Dental Anomalies Part 1 | Garvey et al. 1999; Sasaki et al. 2007; Ferrés-Padró et al. 2009; Kaya et al. 2011 |
| Hyperdontia — true prevalence may reach | 10% (many unerupted) | L2 Dental Anomalies Part 1 | — |
| Hyperdontia — M:F ratio | 1.18:1 (up to 2:1) | L2 Dental Anomalies Part 1 | Brook 1984; Fernandez Montenegro et al. 2006; Liu et al. 2007 |
| Dentigerous cyst risk from supernumerary | ~10% (3rd–4th decade) | L2 Dental Anomalies Part 1 | — |
| Recommended supernumerary removal age | Before age 7 | L2 Dental Anomalies Part 1 | Omer, 2010 |
| Hypodontia (def.) / Oligodontia (def.) | <6 / ≥6 missing teeth | L2 Dental Anomalies Part 1 | — |
| Hypodontia severity — mild/moderate/severe | 1–2 / 3–5 / ≥6 teeth | L2 Dental Anomalies Part 1 | Goodman 1994; Dhanrajjani 2002; Jones 2009 |
| WA referral to Perth Children’s Hospital | ≥10 missing permanent (excl. 3rd molars) | L2 Dental Anomalies Part 1 | — |
| Hypodontia — primary / permanent (Caucasian) | 0.1–4.1% / 4–6% | L2 Dental Anomalies Part 1 | Saito 1959; Brook 1974; Tongkoom 1994; Niswander & Sujaku 1983; Tsai 1996 |
| Hypodontia — F:M ratio (permanent) | 3:2 | L2 Dental Anomalies Part 1 | Nunn 2003; Larmour 2005 |
| Hypodontia — maxilla / mandible distribution | 53.2% / 46.8% | L2 Dental Anomalies Part 1 | — |
| Most-common missing teeth — mand. 2nd premolar / max. lateral incisor / max. 2nd premolar | 29.9% / 24.3% / 13.7% | L2 Dental Anomalies Part 1 | Khalaf et al. 2014 |
| Hypodontia — a major feature of | >50 syndromes | L2 Dental Anomalies Part 1 | — |
| Microdontia — primary / permanent | 0.5–6.3% / 1.9–6.9% | L2 Dental Anomalies Part 1 | Brook 1974; Ooshima et al. 1996; Tsai 1996; Aldred et al. 2012 |
| Macrodontia — threshold / primary / permanent | 1 mm larger / 0–2.3% / 1.1–3.6% | L2 Dental Anomalies Part 1 | Chaudhary et al. 1997; Brook 1974; Tsai 1996 |
Tooth-shape anomalies (prevalence)
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Double tooth — primary / permanent | 0.1–3.0% / 0.1–0.8% | L4 Dental Anomalies, L4 Dental Anomalies 2 | Brook 1974; Buenviaje 1984; Cheng 2003; Sekerci et al. 2011; Chung et al. 1972; Tsai 1996 |
| Talon cusp — primary / permanent | 0.5–0.6% / 1–2.5% | L4 Dental Anomalies, L4 Dental Anomalies 2 | Chen 1986; Ooshima et al. 1996; Liu 1996; Mavrodisz et al. 2003; King et al. 2010 |
| Talon cusp — M:F ratio | 1.9:1 | L4 Dental Anomalies, L4 Dental Anomalies 2 | Lee et al. 2007 |
| Talon cusp on primary max. incisors — prevalence / associated with other anomalies / supernumerary elsewhere | ~3% / 80% / 60% | L4 Dental Anomalies, L4 Dental Anomalies 2 | — |
| Talon-cusp gradual reduction | Over 2–3 appointments (tertiary dentine between) | L4 Dental Anomalies, L4 Dental Anomalies 2 | — |
| Lee 2007 — central incisor successors with no abnormality / lateral incisor cases with abnormality | 32/35 (91.4%) / 18/23 (78.3%) | L4 Dental Anomalies, L4 Dental Anomalies 2 | Lee CK et al., Int J Paediatr Dent. 2007 |
| Dens evaginatus — prevalence / most-commonly identified age | 0.1–4.7% / 10–13 yrs | L4 Dental Anomalies, L4 Dental Anomalies 2 | — |
| Dens evaginatus — Oehlers pulp content (wide/narrow/constricted/isolated/none) | 34% / 22% / 14% / 20% / 10% | L4 Dental Anomalies, L4 Dental Anomalies 2 | Oehlers, 1967 |
| Dens invaginatus — primary / permanent | 0.1% / 0.2–10.0% | L4 Dental Anomalies, L4 Dental Anomalies 2 | Brook, 1974 |
Tooth anomaly — most-affected tooth & dentition
Quick rule
Number & size anomalies (hyperdontia, hypodontia, microdontia, macrodontia) are all more common in the permanent dentition. Among shape anomalies, only double tooth favours primary; talon cusp, dens evaginatus & dens invaginatus favour permanent.
| Anomaly | Most-likely affected tooth | More common in | Source | Paper |
|---|---|---|---|---|
| Hyperdontia (supernumerary) | Mesiodens (anterior maxillary midline) — most common type; region order: maxillary incisor > mandibular premolar > maxillary molar | Permanent (0.5–3.5% vs 0.2–0.8%) | L2 Dental Anomalies Part 1 | Grahnen & Lindahl 1961; King et al. 1993 |
| Hypodontia | Mandibular 2nd premolar (29.9% — most common overall), then maxillary lateral incisor (24.3%) & 3rd molars (“last tooth in each field”, Butler); maxilla 53.2% > mandible 46.8% | Permanent (4–6% Caucasian vs 0.1–4.1%) | L2 Dental Anomalies Part 1 | Khalaf et al. 2014; Butler 1939 |
| Microdontia | Maxillary lateral incisor (peg lateral); maxillary > mandibular | Permanent (1.9–6.9% vs 0.5–6.3%) | L2 Dental Anomalies Part 1 | Brook 1974; Ooshima et al. 1996 |
| Macrodontia | Incisors & canines (isolated, most frequent); also mandibular premolars/molars & 3rd molars | Permanent (1.1–3.6% vs 0–2.3%) | L2 Dental Anomalies Part 1 | Brook 1974; Tsai 1996 |
| Double tooth (umbrella) | Incisor & canine region; more common in maxilla | Primary (0.1–3.0% vs 0.1–0.8%) | L4 Dental Anomalies, L4 Dental Anomalies 2 | Brook 1974; Buenviaje 1984; Cheng 2003 |
| → Fusion | Incisors/canines, maxilla (reduced tooth count in quadrant) | Primary | L4 Dental Anomalies, L4 Dental Anomalies 2 | — |
| → Gemination | Maxilla; presents as a macrodont in permanent (normal tooth count) | Primary | L4 Dental Anomalies, L4 Dental Anomalies 2 | Ravn 1971; Kelly 1978 |
| Concrescence | Union at root/cementum level (specific tooth not stated; classically maxillary molars) | Not specified in lecture | L4 Dental Anomalies | — |
| Talon cusp | Maxillary anterior — incisors (esp. lateral) & canines, palatal surface | Permanent (1–2.5% vs 0.5–0.6%) | L4 Dental Anomalies, L4 Dental Anomalies 2 | Ooshima et al. 1996; King et al. 2010 |
| Dens evaginatus | Premolars (rarely canines/molars); marked mandibular predominance; bilateral | Permanent (“mostly permanent teeth”) | L4 Dental Anomalies, L4 Dental Anomalies 2 | Lau 1955; Oehlers 1967 |
| Dens invaginatus | Maxillary lateral incisor (most common); primary: canine, max. central incisor, mand. 2nd molar | Permanent (0.2–10.0% vs 0.1%) | L4 Dental Anomalies, L4 Dental Anomalies 2 | Brook 1974 |
MIH / HSPM — composition & burden (non-prevalence)
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Age for accurate MIH assessment | 6–7 yrs (all FPMs + incisors erupted) | L5 MIH and HSPM | — |
| Protein content — brown enamel vs sound | 15–21× higher | L5 MIH and HSPM | — |
| Protein content — white/yellow enamel vs sound | ~8× higher | L5 MIH and HSPM | — |
| Transverse microradiography density — brown / yellow / white / unaffected | 1.79 / 2.21 / 2.43 / 2.46 g/cm³ | L5 MIH and HSPM | Eur Arch Paediatr Dent. 2017 |
| MIH molars affected (range) | 1 to 4 | L5 MIH and HSPM | — |
| By age 18, MIH patients underwent FPM treatment | 4.2× more often than controls | L5 MIH and HSPM | Jälevik & Klingberg 2012 |
| Restorative burden avoided by timely extraction | 50–60 yrs of operative cycles | L5 MIH and HSPM | — |
| Microabrasion — cycles / time per tooth per cycle | 2–3 cycles / 30–60 s | L5 MIH and HSPM | — |
| Early referral for moderate/severe molar defects | Before 8 yrs | L5 MIH and HSPM | — |
Orofacial infection epidemiology & disease course
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| HSV — proportion of population infected / manifesting acute primary symptoms | ~60% / ~1% | L7 Orofacial Infections In Paediatric Dentistry | — |
| Primary herpetic gingivostomatitis — onset / peak incidence | After 6 months / 12–18 months | L7 Orofacial Infections In Paediatric Dentistry | — |
| Herpetic gingivostomatitis — incubation / prodrome / ulcer size / fever / healing | 3–5 days / 48 h / 3 mm / 37.8–38.9 °C / 10–14 days | L7 Orofacial Infections In Paediatric Dentistry | — |
| Herpes labialis — erythema before lesions / crusting / healing | 6–24 h / within 2 days / 7–10 days | L7 Orofacial Infections In Paediatric Dentistry | — |
| Infectious mononucleosis — common age / EBV chronic fatigue | 1–5 yrs / <10% of cases | L7 Orofacial Infections In Paediatric Dentistry | — |
| Herpangina — age / incubation / lesion count / ulcer size / healing | ≤10 yrs / 4–7 days / 2–6 / 2–4 mm / 1–2 wks | L7 Orofacial Infections In Paediatric Dentistry | — |
| Hand, foot & mouth — age / oral lesion count / ulcer size / course | ≤10 yrs / 1–30 / 2–4 mm / 7–10 days | L7 Orofacial Infections In Paediatric Dentistry | — |
| Varicella — incubation (avg) / immunised course / oral vesicle → ulcer size | 10–21 days (15) / 4–6 days / 3–4 mm → 1–3 mm | L7 Orofacial Infections In Paediatric Dentistry | — |
| Varicella oral severity — mild / severe | 1–2 ulcers (1–3 days) / up to 30 ulcers (5–10 days) | L7 Orofacial Infections In Paediatric Dentistry | — |
Dental trauma (primary dentition) — epidemiology
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Head/facial injuries as proportion of all somatic injuries (preschool) | 40% | L8 Dental Trauma In the Primary Dentition | Glendor et al. 1996 |
| Toddler fall frequency | 17×/hour | L8 Dental Trauma In the Primary Dentition | Adolph et al. 2012 |
| Children sustaining injuries to primary teeth | 30% | L8 Dental Trauma In the Primary Dentition | — |
| Children with history of traumatic dental injuries | 46% | L8 Dental Trauma In the Primary Dentition | Andreasen & Ravn 1972 |
| Primary-tooth injuries that are concussion/subluxation/luxation | ~80% (softer alveolar bone) | L8 Dental Trauma In the Primary Dentition | Heinz |
| Post-trauma pulp necrosis / discoloration | ~25% / ~52% | L8 Dental Trauma In the Primary Dentition | — |
| Colour change as most common complication overall | ~50% (not always = necrosis) | L8 Dental Trauma In the Primary Dentition | — |
Dental trauma — management thresholds & timings
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Steep occlusal radiograph angulation | 65° | L8 Dental Trauma In the Primary Dentition | — |
| Recommended incisor trauma series | 1 steep occlusal + 3 periapical (bisecting angle) | L8 Dental Trauma In the Primary Dentition | — |
| Concussion/subluxation — complication risk / timeline | <10% / most within first 12 months | L8 Dental Trauma In the Primary Dentition | — |
| Recall — concussion/subluxation | 1 wk, 1 mo, 3 mo, 6 mo, 1 yr | L8 Dental Trauma In the Primary Dentition | — |
| Higher complication risk above age | >4 yrs | L8 Dental Trauma In the Primary Dentition | Lauridsen et al. 2017 |
| Intrusion — spontaneous re-eruption / even if / complication rate | 80% / 100% intruded / ~⅓ | L8 Dental Trauma In the Primary Dentition | — |
| Lowest pulp-necrosis risk after intrusion | <2 yrs | L8 Dental Trauma In the Primary Dentition | Lauridsen et al. 2017 |
| Monitoring — intrusion | 1 wk, 1 mo, 3, 6, 9, 12 mo | L8 Dental Trauma In the Primary Dentition | — |
| Complication rate — extrusion / lateral luxation | 15–43% / 20–40% | L8 Dental Trauma In the Primary Dentition | — |
| Extrusion/lateral-luxation complications within first 12 months | 96% | L8 Dental Trauma In the Primary Dentition | Lauridsen et al. 2017 |
Borum & Andreasen (387 traumatised primary teeth) — outcomes by luxation type: colour change extrusion 80.0% / lateral luxation 62.3% · pulp necrosis intrusion 37.6% / extrusion 30.0% · pulp canal obliteration extrusion 70.0% / lateral luxation 41.8% · inflammatory resorption intrusion 14.1% / lateral luxation 13.0% · permanent displacement intrusion 17.6% · no complications concussion 57.1% / subluxation 43.2% / extrusion 0%. — L8 Dental Trauma In the Primary Dentition (Borum & Andreasen)
Eruption & developmental timings
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Second primary molars erupt | ~20–30 months | W1 ICDAS and intro to caries | — |
| First permanent molar — “in bone” → “in occlusion” | ~1 year | W1 ICDAS and intro to caries | — |
| Primary teeth begin calcifying | 4th fetal month | L5 MIH and HSPM | — |
| Panoramic “canine checks” (mixed dentition) | Ages 9–12 | L1 The Paediatric Clinical examination LMS | — |
Clinical / appointment parameters
| Statistic | Figure | Source | Paper |
|---|---|---|---|
| Max clinical appointment time for children | 30–40 min | L1 The Paediatric Clinical examination LMS | — |
| Patient identifiers to confirm identity | 2–3 | L1 The Paediatric Clinical examination LMS | — |
| Intra-oral film sizes (anterior/occlusal) | 0, 1, 2 (size 0 for young children) | L1 The Paediatric Clinical examination LMS | — |
| Antibiotics-before-extraction delay (non-systemic cases) | ~5 days (peak effect) | W1 ICDAS and intro to caries | — |
Note: the eruption-timing, ~5-day antibiotic-delay, and articaine worked-example figures above are drawn from the W2/W3 workshop case files (
[[W2 Cases_integrated]],[[W3 Case 2_integrated]]).
Sources
- L1 The Paediatric Clinical examination LMS
- L2 Dental Anomalies Part 1
- L4 Dental Anomalies · L4 Dental Anomalies 2
- L5 MIH and HSPM
- L6 Antibiotics LA and Analgesics in Paediatric Dentistry
- L7 Orofacial Infections In Paediatric Dentistry
- L8 Dental Trauma In the Primary Dentition
- W1 ICDAS and intro to caries · W1 Cases
- W2 Cases_integrated
- W3 Case 1_integrated · W3 Case 2_integrated · W3 Case 3_integrated · W3 Extra information
- Related: R1 Antibiotic Selection Table