Pediatric Important Numbers

How to use this note

A quick-reference consolidation of every quantitative fact from the DMD3S1 Pediatric Dentistry lectures and workshops, ordered by exam priority: 1) caries statistics → 2) local anaesthesia → 3) medications → 4) other statistics.

  • Every figure links to its source lecture via wikilink.
  • Where a figure comes from an in-lecture paper, that paper is named in plain text in the Paper column (these are not vault notes). = stated by the lecturer without a cited source.
  • Drug doses cross-reference [[R1 Antibiotic Selection Table]] for penicillin-allergy tiers.

1. Caries Statistics

Highest-yield caries figures

= Primary→permanent caries risk ≈ (RR 2.6). MIH ≈ 1 in 7 children; HSPM ≈ 1 in 10.

Caries risk, detection & ICDAS

StatisticFigureSource LectureIn-lecture Paper
Moderate caries-risk threshold1–2 new lesions/yearL1 The Paediatric Clinical examination LMS
High caries-risk threshold≥3 new lesions/yearL1 The Paediatric Clinical examination LMS
Bitewing = gold standard for caries; film size for young childSize 0L1 The Paediatric Clinical examination LMS
Occlusal lesions detected by bitewing (sensitivity)~33%W1 ICDAS and intro to caries
ICDAS second-digit severity range0–6W1 ICDAS and intro to caries
ICDAS codes favouring preventive/microinvasive care0–4W1 ICDAS and intro to caries
ICDAS codes indicating operative treatment5–6W1 ICDAS and intro to caries
ICDAS threshold used in epidemiological/field surveys3+W1 ICDAS and intro to caries

ICDAS code key (second digit): 0 sound · 1 first visual change (after drying / in pit-fissure) · 2 distinct change visible when wet · 3 localised enamel breakdown / microcavitation, no dentine · 4 dark dentine shadow ± enamel breakdown · 5 distinct cavity, dentine visible, <½ surface · 6 extensive cavity, dentine visible, >½ surface. — W1 ICDAS and intro to caries

Fluoride

StatisticFigureSource LectureIn-lecture Paper
Recommended fluoride toothpaste (prevention & sensitivity)1000 ppmL5 MIH and HSPM, W1 ICDAS and intro to caries
Commercial “3–5 year” children’s pastes (often only)500 ppmW1 ICDAS and intro to caries
Toothbrushing frequencyTwice dailyW1 ICDAS and intro to caries

Caries as a predictor / progression

StatisticFigureSource LectureIn-lecture Paper
Children with MIH — odds of DMFT > 0 vs non-MIH5.89× (95% CI 2.69–12.88; P<0.05)W1 ICDAS and intro to cariesAmericano et al. 2016
Primary tooth with proximal caries → caries on adjacent proximal surface69%W1 ICDAS and intro to cariesDean et al. 1997
Proximal lesion in one quadrant → another primary-molar proximal lesion in a different quadrant89%W1 ICDAS and intro to cariesDean et al. 1997
Caries in primary teeth → relative likelihood of permanent-tooth caries≈3× (RR 2.6; 95% CI 1.4–4.7)W1 ICDAS and intro to cariesLi and Wang 2002
Predictive value of primary-molar caries for permanent-dentition caries85.4% (highest)W1 ICDAS and intro to cariesLi and Wang 2002
↑ risk of mesial caries on first permanent molar from distal caries on second primary molar15×W1 ICDAS and intro to caries

Note: [[W3 Extra information]] (caries-prediction workshop handout) is the in-vault source for the Americano, Dean, and Li & Wang figures above.

MIH prevalence (developmental, caries-relevant)

StatisticFigureSource LectureIn-lecture Paper
MIH prevalence (population-dependent)3% – 25%L5 MIH and HSPM
MIH worldwide range2.4% – 40.2%L5 MIH and HSPMJälevik, 2010
MIH prevalence — Australia22% – 44%L5 MIH and HSPMArrow et al. 2008; Balmer et al. 2005
MIH prevalence — New Zealand14.9% – 18.8%L5 MIH and HSPMMahoney & Morrison, 2009/2011
Pooled global MIH prevalence (meta-analysis, 70 studies)14.2% (≈1 in 7)L5 MIH and HSPMZhao et al. 2018
MIH — males / females14.3% / 14.4% (NS)L5 MIH and HSPMZhao et al. 2018
MIH — ≤10 yrs / >10 yrs15.1% / 12.1%L5 MIH and HSPMZhao et al. 2018
MIH — South America / Spain (highest)18.0% / 21.1%L5 MIH and HSPMZhao et al. 2018
StatisticFigureSource LectureIn-lecture Paper
HSPM global prevalence≈10% (range 5–15%)L5 MIH and HSPM
HSPM prevalence — Australia14%L5 MIH and HSPMOwen et al. 2017
HSPM as predictor of MIH (odds ratio)4.66 (up to 6× in some studies)L5 MIH and HSPM
HSPM–MIH co-occurrence~20%L5 MIH and HSPMGarot et al. 2018
Child with HSPM → ↑ risk of similar permanent-dentition defects2.4 – 10×W1 ICDAS and intro to caries

2. Local Anaesthesia

Highest-yield LA figures

==Lignocaine & Articaine max = 7 mg/kg; Bupivacaine max = 2 mg/kg.== Australian carpule = 2.2 mL. ==2% solution = 20 mg/mL; 4% = 40 mg/mL.==

Agents

AgentConcentrationVasoconstrictorMax doseNotesSourcePaper
Lignocaine2% (= 20 mg/mL)Adrenaline 1:80,0007 mg/kgStandard agent; acidic infected tissue slows onset/shortens actionL6 Antibiotics LA and Analgesics in Paediatric Dentistry
Articaine4% (= 40 mg/mL)Adrenaline 1:100,0007 mg/kgeffective mandibular infiltration, short onset; 1:80,000 better haemostasis than 1:100,000L6 Antibiotics LA and Analgesics in Paediatric Dentistry, W1 ICDAS and intro to caries
Bupivacaine0.5%Adrenaline 1:200,0002 mg/kgLong-acting: ~6 h pulpal, 9–12 h soft tissue → ↑ self-injury risk in childrenL6 Antibiotics LA and Analgesics in Paediatric Dentistry
Topical lignocaine2–5% gel / 5% adhesive vs 10% ointment(verify product)Dwell ~2 min, then wipe with damp gauze; verify concentrationL6 Antibiotics LA and Analgesics in Paediatric Dentistry

Dose calculation & technique

ParameterFigureSourcePaper
Percentage → mg/mL conversion1% = 1 g/100 mL = 10 mg/mL (so 2% = 20 mg/mL, 4% = 40 mg/mL)L6 Antibiotics LA and Analgesics in Paediatric Dentistry
Carpule volume (Australia standard)2.2 mL (intl. often 1.8 mL or 1.7 mL)L6 Antibiotics LA and Analgesics in Paediatric Dentistry
2% lignocaine content per carpule2.2 mL → 44 mg · 1.8 mL → 36 mg · 1.7 mL → 34 mgL6 Antibiotics LA and Analgesics in Paediatric Dentistry
Worked max dose, 25 kg child (2% lignocaine)7 × 25 = 175 mg = 8.75 mL ≈ 3.9 carpulesL6 Antibiotics LA and Analgesics in Paediatric Dentistry
Worked max dose, 18 kg child (4% articaine)7 × 18 = 126 mg = 3.15 mL ≈ 1.4 carpulesW1 ICDAS and intro to caries
IANB depth to bony contact~20 mmL6 Antibiotics LA and Analgesics in Paediatric Dentistry
Needle gauge for IANB27–30 G (short needle in children)L6 Antibiotics LA and Analgesics in Paediatric Dentistry
Staged injection volume½ carpule initially, re-inject only if inadequateL6 Antibiotics LA and Analgesics in Paediatric Dentistry

Note: the 18 kg articaine worked example is sourced from the W3 abscess workshop case ([[W3 Case 2_integrated]]).


3. Medications — Dosages & Timings

Highest-yield drug doses

Penicillin V / Amoxycillin / Cephalexin: 12.5 mg/kg (max 500 mg). Clindamycin 7.5 mg/kg (max 300 mg); Metronidazole 10 mg/kg (max 400 mg). Paracetamol 15 mg/kg (max 4 g/day); Ibuprofen 5–10 mg/kg (max 2400 mg/day).

Antibiotics — odontogenic infection (all oral, 5-day course)

See [[R1 Antibiotic Selection Table]] for the penicillin-allergy tier list.

DrugUsed For (Condition)DoseFrequency / DurationSourcePaper
Phenoxymethylpenicillin (Pen V)Odontogenic infection — drug of first choice12.5 mg/kg up to 500 mg6-hourly × 5 days (1 h before / 2 h after meals)L6 Antibiotics LA and Analgesics in Paediatric Dentistry
AmoxycillinOdontogenic infection12.5 mg/kg up to 500 mg8-hourly × 5 daysL6 Antibiotics LA and Analgesics in Paediatric Dentistry
CephalexinOdontogenic infection (penicillin-sensitive alternative)12.5 mg/kg up to 500 mg8-hourly × 5 daysL6 Antibiotics LA and Analgesics in Paediatric Dentistry
ClindamycinOdontogenic infection — first choice if penicillin-allergic7.5 mg/kg up to 300 mg8-hourly × 5 daysL6 Antibiotics LA and Analgesics in Paediatric Dentistry
MetronidazoleSpreading / anaerobic infection (supplements penicillin or clindamycin)10 mg/kg up to 400 mg12-hourly × 5 days; avoid alcohol 24 h after course (adults)L6 Antibiotics LA and Analgesics in Paediatric Dentistry

Suspension concentrations to specify on the script: Pen V 125 / 150 / 250 mg per 5 mL · Amoxycillin 100 mg/mL, 125 / 250 / 500 mg per 5 mL · Cephalexin 125 / 250 mg per 5 mL · Clindamycin 150 mg caps · Metronidazole 200 mg/5 mL (40 mg/mL). — L6 Antibiotics LA and Analgesics in Paediatric Dentistry

Analgesics

DrugUsed For (Condition)DoseFrequency / DurationSourcePaper
ParacetamolPost-op / dental pain & fever — first line15 mg/kg (max 1 g/dose)4–6-hourly, max 4 g/day; conc. 120 mg/5 mL (24 mg/mL)L6 Antibiotics LA and Analgesics in Paediatric Dentistry
IbuprofenPost-op / dental pain — first line (anti-inflammatory)5–10 mg/kg6–8-hourly, max 2400 mg/day; conc. 100 mg/5 mL; ≤3 days without reviewL6 Antibiotics LA and Analgesics in Paediatric DentistryKent et al. J Am Coll Cardiol. 2018;72(3):255-67

Analgesic age restrictions

  • Codeine: not in children <12 yrs; also contraindicated 12–18 yrs after tonsillectomy/adenoidectomy. Rescheduled to S4 (prescription-only) on 1 Feb 2018. — Pediatrics. 2016;138(4):e20162396
  • Aspirin: banned <16 yrs (Reye syndrome). — Glasgow JF, Drug Saf 2002; Schrör K, BMJ 2002; Macdonald S, BMJ 2002

Orofacial infection medications

DrugUsed For (Condition)DoseFrequency / DurationSourcePaper
Aciclovir (PO)Primary herpetic gingivostomatitis (HSV) — antiviral20 mg/kg5×/day; within first 72–96 h (prodromal phase); limited evidence <2 yrsL7 Orofacial Infections In Paediatric Dentistry
Aciclovir (IV)Severe / immunocompromised HSV10 mg/kgIVL7 Orofacial Infections In Paediatric Dentistry
Chlorhexidine 0.2% (swab)Herpetic gingivostomatitis — severe ulceration, young children0.2% on cotton woolOver affected areasL7 Orofacial Infections In Paediatric Dentistry
Chlorhexidine 0.2% (mouthwash)Herpetic gingivostomatitis — older children0.2%, 10 mLEvery 4 hL7 Orofacial Infections In Paediatric Dentistry
Chlorhexidine gelPost-trauma (concussion/subluxation) oral hygieneFinger/brush applicationFirst week only, then brush alternate daysL8 Dental Trauma In the Primary Dentition
Difflam C (benzydamine + chlorhexidine)Herpetic gingivostomatitis — combination rinseBenzydamine 0.15% + chlorhexidine 0.12%L7 Orofacial Infections In Paediatric Dentistry
Tetracycline / minocycline mouthwashHerpetic gingivostomatitis — children >12 yrsL7 Orofacial Infections In Paediatric Dentistry
Varicella antivirals (aciclovir/valaciclovir/famciclovir)Varicella (chickenpox)Most effective within 24 h of rash onsetL7 Orofacial Infections In Paediatric Dentistry
ParacetamolHerpetic gingivostomatitis — pain/fever15 mg/kgEvery 4–6 hL7 Orofacial Infections In Paediatric Dentistry

Topical anaesthetic cautions (infection setting)


4. Other Statistics

Tooth-number & size anomalies (prevalence)

StatisticFigureSourcePaper
Hyperdontia — primary / permanent dentition0.2–0.8% / 0.5–3.5%L2 Dental Anomalies Part 1Garvey et al. 1999; Sasaki et al. 2007; Ferrés-Padró et al. 2009; Kaya et al. 2011
Hyperdontia — true prevalence may reach10% (many unerupted)L2 Dental Anomalies Part 1
Hyperdontia — M:F ratio1.18:1 (up to 2:1)L2 Dental Anomalies Part 1Brook 1984; Fernandez Montenegro et al. 2006; Liu et al. 2007
Dentigerous cyst risk from supernumerary~10% (3rd–4th decade)L2 Dental Anomalies Part 1
Recommended supernumerary removal ageBefore age 7L2 Dental Anomalies Part 1Omer, 2010
Hypodontia (def.) / Oligodontia (def.)<6 / ≥6 missing teethL2 Dental Anomalies Part 1
Hypodontia severity — mild/moderate/severe1–2 / 3–5 / ≥6 teethL2 Dental Anomalies Part 1Goodman 1994; Dhanrajjani 2002; Jones 2009
WA referral to Perth Children’s Hospital≥10 missing permanent (excl. 3rd molars)L2 Dental Anomalies Part 1
Hypodontia — primary / permanent (Caucasian)0.1–4.1% / 4–6%L2 Dental Anomalies Part 1Saito 1959; Brook 1974; Tongkoom 1994; Niswander & Sujaku 1983; Tsai 1996
Hypodontia — F:M ratio (permanent)3:2L2 Dental Anomalies Part 1Nunn 2003; Larmour 2005
Hypodontia — maxilla / mandible distribution53.2% / 46.8%L2 Dental Anomalies Part 1
Most-common missing teeth — mand. 2nd premolar / max. lateral incisor / max. 2nd premolar29.9% / 24.3% / 13.7%L2 Dental Anomalies Part 1Khalaf et al. 2014
Hypodontia — a major feature of>50 syndromesL2 Dental Anomalies Part 1
Microdontia — primary / permanent0.5–6.3% / 1.9–6.9%L2 Dental Anomalies Part 1Brook 1974; Ooshima et al. 1996; Tsai 1996; Aldred et al. 2012
Macrodontia — threshold / primary / permanent1 mm larger / 0–2.3% / 1.1–3.6%L2 Dental Anomalies Part 1Chaudhary et al. 1997; Brook 1974; Tsai 1996

Tooth-shape anomalies (prevalence)

StatisticFigureSourcePaper
Double tooth — primary / permanent0.1–3.0% / 0.1–0.8%L4 Dental Anomalies, L4 Dental Anomalies 2Brook 1974; Buenviaje 1984; Cheng 2003; Sekerci et al. 2011; Chung et al. 1972; Tsai 1996
Talon cusp — primary / permanent0.5–0.6% / 1–2.5%L4 Dental Anomalies, L4 Dental Anomalies 2Chen 1986; Ooshima et al. 1996; Liu 1996; Mavrodisz et al. 2003; King et al. 2010
Talon cusp — M:F ratio1.9:1L4 Dental Anomalies, L4 Dental Anomalies 2Lee et al. 2007
Talon cusp on primary max. incisors — prevalence / associated with other anomalies / supernumerary elsewhere~3% / 80% / 60%L4 Dental Anomalies, L4 Dental Anomalies 2
Talon-cusp gradual reductionOver 2–3 appointments (tertiary dentine between)L4 Dental Anomalies, L4 Dental Anomalies 2
Lee 2007 — central incisor successors with no abnormality / lateral incisor cases with abnormality32/35 (91.4%) / 18/23 (78.3%)L4 Dental Anomalies, L4 Dental Anomalies 2Lee CK et al., Int J Paediatr Dent. 2007
Dens evaginatus — prevalence / most-commonly identified age0.1–4.7% / 10–13 yrsL4 Dental Anomalies, L4 Dental Anomalies 2
Dens evaginatus — Oehlers pulp content (wide/narrow/constricted/isolated/none)34% / 22% / 14% / 20% / 10%L4 Dental Anomalies, L4 Dental Anomalies 2Oehlers, 1967
Dens invaginatus — primary / permanent0.1% / 0.2–10.0%L4 Dental Anomalies, L4 Dental Anomalies 2Brook, 1974

Tooth anomaly — most-affected tooth & dentition

Quick rule

Number & size anomalies (hyperdontia, hypodontia, microdontia, macrodontia) are all more common in the permanent dentition. Among shape anomalies, only double tooth favours primary; talon cusp, dens evaginatus & dens invaginatus favour permanent.

AnomalyMost-likely affected toothMore common inSourcePaper
Hyperdontia (supernumerary)Mesiodens (anterior maxillary midline) — most common type; region order: maxillary incisor > mandibular premolar > maxillary molarPermanent (0.5–3.5% vs 0.2–0.8%)L2 Dental Anomalies Part 1Grahnen & Lindahl 1961; King et al. 1993
HypodontiaMandibular 2nd premolar (29.9% — most common overall), then maxillary lateral incisor (24.3%) & 3rd molars (“last tooth in each field”, Butler); maxilla 53.2% > mandible 46.8%Permanent (4–6% Caucasian vs 0.1–4.1%)L2 Dental Anomalies Part 1Khalaf et al. 2014; Butler 1939
MicrodontiaMaxillary lateral incisor (peg lateral); maxillary > mandibularPermanent (1.9–6.9% vs 0.5–6.3%)L2 Dental Anomalies Part 1Brook 1974; Ooshima et al. 1996
MacrodontiaIncisors & canines (isolated, most frequent); also mandibular premolars/molars & 3rd molarsPermanent (1.1–3.6% vs 0–2.3%)L2 Dental Anomalies Part 1Brook 1974; Tsai 1996
Double tooth (umbrella)Incisor & canine region; more common in maxillaPrimary (0.1–3.0% vs 0.1–0.8%)L4 Dental Anomalies, L4 Dental Anomalies 2Brook 1974; Buenviaje 1984; Cheng 2003
FusionIncisors/canines, maxilla (reduced tooth count in quadrant)PrimaryL4 Dental Anomalies, L4 Dental Anomalies 2
GeminationMaxilla; presents as a macrodont in permanent (normal tooth count)PrimaryL4 Dental Anomalies, L4 Dental Anomalies 2Ravn 1971; Kelly 1978
ConcrescenceUnion at root/cementum level (specific tooth not stated; classically maxillary molars)Not specified in lectureL4 Dental Anomalies
Talon cuspMaxillary anterior — incisors (esp. lateral) & canines, palatal surfacePermanent (1–2.5% vs 0.5–0.6%)L4 Dental Anomalies, L4 Dental Anomalies 2Ooshima et al. 1996; King et al. 2010
Dens evaginatusPremolars (rarely canines/molars); marked mandibular predominance; bilateralPermanent (“mostly permanent teeth”)L4 Dental Anomalies, L4 Dental Anomalies 2Lau 1955; Oehlers 1967
Dens invaginatusMaxillary lateral incisor (most common); primary: canine, max. central incisor, mand. 2nd molarPermanent (0.2–10.0% vs 0.1%)L4 Dental Anomalies, L4 Dental Anomalies 2Brook 1974

MIH / HSPM — composition & burden (non-prevalence)

StatisticFigureSourcePaper
Age for accurate MIH assessment6–7 yrs (all FPMs + incisors erupted)L5 MIH and HSPM
Protein content — brown enamel vs sound15–21× higherL5 MIH and HSPM
Protein content — white/yellow enamel vs sound~8× higherL5 MIH and HSPM
Transverse microradiography density — brown / yellow / white / unaffected1.79 / 2.21 / 2.43 / 2.46 g/cm³L5 MIH and HSPMEur Arch Paediatr Dent. 2017
MIH molars affected (range)1 to 4L5 MIH and HSPM
By age 18, MIH patients underwent FPM treatment4.2× more often than controlsL5 MIH and HSPMJälevik & Klingberg 2012
Restorative burden avoided by timely extraction50–60 yrs of operative cyclesL5 MIH and HSPM
Microabrasion — cycles / time per tooth per cycle2–3 cycles / 30–60 sL5 MIH and HSPM
Early referral for moderate/severe molar defectsBefore 8 yrsL5 MIH and HSPM

Orofacial infection epidemiology & disease course

StatisticFigureSourcePaper
HSV — proportion of population infected / manifesting acute primary symptoms~60% / ~1%L7 Orofacial Infections In Paediatric Dentistry
Primary herpetic gingivostomatitis — onset / peak incidenceAfter 6 months / 12–18 monthsL7 Orofacial Infections In Paediatric Dentistry
Herpetic gingivostomatitis — incubation / prodrome / ulcer size / fever / healing3–5 days / 48 h / 3 mm / 37.8–38.9 °C / 10–14 daysL7 Orofacial Infections In Paediatric Dentistry
Herpes labialis — erythema before lesions / crusting / healing6–24 h / within 2 days / 7–10 daysL7 Orofacial Infections In Paediatric Dentistry
Infectious mononucleosis — common age / EBV chronic fatigue1–5 yrs / <10% of casesL7 Orofacial Infections In Paediatric Dentistry
Herpangina — age / incubation / lesion count / ulcer size / healing≤10 yrs / 4–7 days / 2–6 / 2–4 mm / 1–2 wksL7 Orofacial Infections In Paediatric Dentistry
Hand, foot & mouth — age / oral lesion count / ulcer size / course≤10 yrs / 1–30 / 2–4 mm / 7–10 daysL7 Orofacial Infections In Paediatric Dentistry
Varicella — incubation (avg) / immunised course / oral vesicle → ulcer size10–21 days (15) / 4–6 days / 3–4 mm → 1–3 mmL7 Orofacial Infections In Paediatric Dentistry
Varicella oral severity — mild / severe1–2 ulcers (1–3 days) / up to 30 ulcers (5–10 days)L7 Orofacial Infections In Paediatric Dentistry

Dental trauma (primary dentition) — epidemiology

StatisticFigureSourcePaper
Head/facial injuries as proportion of all somatic injuries (preschool)40%L8 Dental Trauma In the Primary DentitionGlendor et al. 1996
Toddler fall frequency17×/hourL8 Dental Trauma In the Primary DentitionAdolph et al. 2012
Children sustaining injuries to primary teeth30%L8 Dental Trauma In the Primary Dentition
Children with history of traumatic dental injuries46%L8 Dental Trauma In the Primary DentitionAndreasen & Ravn 1972
Primary-tooth injuries that are concussion/subluxation/luxation~80% (softer alveolar bone)L8 Dental Trauma In the Primary DentitionHeinz
Post-trauma pulp necrosis / discoloration~25% / ~52%L8 Dental Trauma In the Primary Dentition
Colour change as most common complication overall~50% (not always = necrosis)L8 Dental Trauma In the Primary Dentition

Dental trauma — management thresholds & timings

StatisticFigureSourcePaper
Steep occlusal radiograph angulation65°L8 Dental Trauma In the Primary Dentition
Recommended incisor trauma series1 steep occlusal + 3 periapical (bisecting angle)L8 Dental Trauma In the Primary Dentition
Concussion/subluxation — complication risk / timeline<10% / most within first 12 monthsL8 Dental Trauma In the Primary Dentition
Recall — concussion/subluxation1 wk, 1 mo, 3 mo, 6 mo, 1 yrL8 Dental Trauma In the Primary Dentition
Higher complication risk above age>4 yrsL8 Dental Trauma In the Primary DentitionLauridsen et al. 2017
Intrusion — spontaneous re-eruption / even if / complication rate80% / 100% intruded / ~⅓L8 Dental Trauma In the Primary Dentition
Lowest pulp-necrosis risk after intrusion<2 yrsL8 Dental Trauma In the Primary DentitionLauridsen et al. 2017
Monitoring — intrusion1 wk, 1 mo, 3, 6, 9, 12 moL8 Dental Trauma In the Primary Dentition
Complication rate — extrusion / lateral luxation15–43% / 20–40%L8 Dental Trauma In the Primary Dentition
Extrusion/lateral-luxation complications within first 12 months96%L8 Dental Trauma In the Primary DentitionLauridsen et al. 2017

Borum & Andreasen (387 traumatised primary teeth) — outcomes by luxation type: colour change extrusion 80.0% / lateral luxation 62.3% · pulp necrosis intrusion 37.6% / extrusion 30.0% · pulp canal obliteration extrusion 70.0% / lateral luxation 41.8% · inflammatory resorption intrusion 14.1% / lateral luxation 13.0% · permanent displacement intrusion 17.6% · no complications concussion 57.1% / subluxation 43.2% / extrusion 0%. — L8 Dental Trauma In the Primary Dentition (Borum & Andreasen)

Eruption & developmental timings

StatisticFigureSourcePaper
Second primary molars erupt~20–30 monthsW1 ICDAS and intro to caries
First permanent molar — “in bone” → “in occlusion”~1 yearW1 ICDAS and intro to caries
Primary teeth begin calcifying4th fetal monthL5 MIH and HSPM
Panoramic “canine checks” (mixed dentition)Ages 9–12L1 The Paediatric Clinical examination LMS

Clinical / appointment parameters

StatisticFigureSourcePaper
Max clinical appointment time for children30–40 minL1 The Paediatric Clinical examination LMS
Patient identifiers to confirm identity2–3L1 The Paediatric Clinical examination LMS
Intra-oral film sizes (anterior/occlusal)0, 1, 2 (size 0 for young children)L1 The Paediatric Clinical examination LMS
Antibiotics-before-extraction delay (non-systemic cases)~5 days (peak effect)W1 ICDAS and intro to caries

Note: the eruption-timing, ~5-day antibiotic-delay, and articaine worked-example figures above are drawn from the W2/W3 workshop case files ([[W2 Cases_integrated]], [[W3 Case 2_integrated]]).


Sources